Neonatal asphyxia per partum can be complicated by severe neurologic sequelae and can lead to neonatal death. Of the 0.2% of live births to cerebral palsy, 10 to 28% would be secondary to neonatal acidosis. Only metabolic acidosis plays a neurotoxic role, explaining the recent interest of Racinet et al. in the development of a new biochemical marker, more specific than pH or base deficit, of neonatal asphyxia per partum at risk of anoxo-ischemic encephalopathy. This eucapnic neonatal pH raises the hope of a biochemical marker of situations at risk of poor prognosis, with high diagnostic value, prognostic and forensic.

Our hypothesis is that eucapnic pH is more efficient than cord blood arterial pH and base deficit in the prediction of adverse neurologic outcomes.

Inclusion Criteria:

• All birth more than 37 weeks of amenorrhea at the maternity ward of the hospital Femme-Mère-Enfant
• from 1st of january 2000 to 31 december 2016

Exclusion Criteria:

• Infants born out of the hospital and secondarily hospitalized in the hospital Femme-Mère-Enfant
• Children with congenital anomalies or without valid arterial and venous umbilical cord samples

All birth more than 37 weeks of amenorrhea at the maternity ward of the hospital Femme-Mère-Enfant from 1st of january 2000 to 31 december 2016