The pathophysiology of Traumatic Optic Neuropathy (TON) include a primary and secondary mechanism of injury. At present, no studies validate a particular approach to the management of TON. There are three management lines for these patients that include 1)observation only;2)medical treatment with high or megadoses of methylprednisolone; and 3)surgical intervention. Studies have shown that forces applied to the frontal bone and malar eminences are transferred and concentrated in the area near the optic canal. The tight adherence of the optic nerve's dural sheath to the periosteum within the optic canal is also thought to contribute to this segment of the nerve being extremely susceptible to the deformative stresses of the skull bones. In this study, investigators aim to make a randomized controlled trial to certify the efficiency of optic nerve canal decompression for TON patients.

surgical intervention's efficiency and outcome for TON patients will be clearly present after this randomized controlled trial study.

Inclusion Criteria:

• Patients with the history of Traumatic Brain Injury or Craniofacial Trauma
• Optic nerve compression（intraneural edema, nerve sheath hematoma or canal fracture with impingement)
• Amplitude and latency abnormal or Ratio of amplitude of abnormal to normal side.

Exclusion Criteria:

• Glasgow Coma Scale，Score<8
• Patients with good Visual Acuity and Visual Evoked Potential amplitude ratio more than 50%
• Other Contraindications for surgery